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Level 36

CVRR 09 (II)


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Arrhythmias
A build-up of local metabolites in cardiac tissue due to ischaemia can cause [_______]
Stable
A type of angina which is intermittent and does not involve death of tissue
Unstable
A type of angina where the plaque has ruptured and is partially blocking a coronary vessel
STEMI
A type of acute coronary syndrome characterised by severe prolonged cardiac pain. ECG presents with ST elevation
NSTEMI
A type of acute coronary syndrome that is identical to unstable angina (partial blockage of coronary vessel) except that actual cell damage is occurring
T wave
An area on an ECG that becomes inverted in the days following a STEMI
Q wave
An area on an ECG that becomes more pronounced in the weeks following a STEMI
Stable angina
ST depression can be indicative of [_______]
20-30
Prolonged cardiac ischemia [_-_] mins causes irreversible cellular damage or necrosis
Oxygen, morphine, nitrates, thrombolytic agents and antiplatelets
The typical acute treatment plan following a STEMI. [_______], [_______], [_______], [_______] and [_______]
ACE inhibitors, Beta blockers, anticoagulants
The typical long term treatment plan following a STEMI/myocardial infarction. [_______], [_______], [_______], nitrates, antiarrhythmics and statins
Nitrates, antiarrhythmics and statins
The typical long term treatment plan following a STEMI/myocardial infarction. ACE inhibitors, Beta blockers, anticoagulants, [_______], [_______] and [_______]
Beta blockers
All drugs ending in 'lol' are [_______]
Oliguria
A term used to describe a low output of urine. This typically accompanies a STEMI
Na+ channel blockers
A Vaughan-Williams class of drugs that reduce phase 0 slope and peak of action potential
Beta blockers
A Vaughan-Williams class of drugs that block sympathetic activity; reduce rate and conduction
K+ channel blockers
A Vaughan-Williams class of drugs that delay repolarization (phase 3) and thereby increase action potential duration and effective refractory period
Ca2+ channel blockers
A Vaughan-Williams class of drugs that block L-type calcium-channels; most effective at SA and AV nodes; reduce rate and conduction